BEGIN:VCALENDAR
VERSION:2.0
PRODID:www.dresden-science-calendar.de
METHOD:PUBLISH
CALSCALE:GREGORIAN
X-MICROSOFT-CALSCALE:GREGORIAN
X-WR-TIMEZONE:Europe/Berlin
BEGIN:VTIMEZONE
TZID:Europe/Berlin
X-LIC-LOCATION:Europe/Berlin
BEGIN:DAYLIGHT
TZNAME:CEST
TZOFFSETFROM:+0100
TZOFFSETTO:+0200
DTSTART:19810329T030000
RRULE:FREQ=YEARLY;INTERVAL=1;BYMONTH=3;BYDAY=-1SU
END:DAYLIGHT
BEGIN:STANDARD
TZNAME:CET
TZOFFSETFROM:+0200
TZOFFSETTO:+0100
DTSTART:19961027T030000
RRULE:FREQ=YEARLY;INTERVAL=1;BYMONTH=10;BYDAY=-1SU
END:STANDARD
END:VTIMEZONE
BEGIN:VEVENT
UID:DSC-22568
DTSTART;TZID=Europe/Berlin:20260205T130000
SEQUENCE:1770273418
TRANSP:OPAQUE
DTEND;TZID=Europe/Berlin:20260205T140000
URL:https://dresden-science-calendar.org/calendar/en/detail/22568
LOCATION:TUD CRTD\, Fetscherstraße 10501307 Dresden
SUMMARY:CMCB Life Sciences Seminar: Prof. Dr. Antje Grosche\, Ludwig-Maximi
 lians-University Munich\, Dep. of Physiological Genomics - BioMedical Cent
 er - BMC
CLASS:PUBLIC
DESCRIPTION:Speaker: \nInstitute of Speaker: \nTopics:\nBiologie\, Willkomm
 en\n Location:\n  Name: TUD CRTD ()\n  Street: Fetscherstraße 105\n  City
 : 01307 Dresden\n  Phone: +49 (0)351 458 82052\n  Fax: +49 (0)351 458 8205
 9 \nDescription: <p><strong>Host: </strong>Marius Ader (CRTD)</p> <p><stro
 ng>Title: </strong>\"Focus on glia cells: New gene therapy approaches for 
 retinal diseases\"</p> <p><strong>Abstract: </strong>Our research aims to 
 explore the intricate interactions between Müller cells\, microglia and n
 eurons in both healthy and diseased retinas. We are particularly intereste
 d in understanding the effects of AAV-vectored gene therapies targeting th
 ese interactions\, in particular the coordination of the retinal immune re
 sponse\, for example by modulating retinal complement homeostasis or gluco
 corticoid receptor signaling. We demonstrate that our approach of deliveri
 ng therapeutic gene expression to Müller cells can improve disease progre
 ssion in models of diabetic retinopathy or ischemic stress\, independent o
 f the underlying cause of the disease\, and thus may benefit patients who 
 are not candidates for currently available treatment strategies\, since mo
 st\, if not all\, retinal diseases involve Müller cell gliosis and inflam
 matory processes that potentially accelerate neuronal dysfunction.</p>
DTSTAMP:20260416T085138Z
CREATED:20260108T063716Z
LAST-MODIFIED:20260205T063658Z
END:VEVENT
END:VCALENDAR